1956, a colony of chimps was noted to be suffering from colds.

This lead to the naming of a new virus called chimpanzee coyza agent. Over time this idea has been beaten up, debated, put under a microscope and rechristened respiratory syncytial virus (RSV) - derived from the type of disease the virus creates in humans.

RSV, that ubiquitous agent present worldwide. It is highly contagious for people of all ages. RSV in tissue culture produces an amalgamation of cells that have lost their borders, called syncytia, and appear to be glistening, amoeboid islands in a sea of uninfected healthy cells.

The virus creates a spectrum of illness and much of its inner personality still remains enigmatic. We call the RSV the winter virus becasue of its distinctive epidemiological pattern.

Immunity to RSV still remains a conundrum, with past infection only conferring transient protection. The presence of antibody in the blood, the proteins produced by the body against specific infections agents, is usually indicative of ones immunity to that agent. And we know that All adults have specific serum antibody against RSV from previous infections (most are infected by age 3), furthermore all infants possess specific RSV antibody transferred from their mothers at birth.

Despite RSV antibodies (and counter to popular belief), the presence or amount of such antibody does not corelate with resistance to RSV infection.

This is in contrast to most other such viral agents. Medicine is still not clear about what factors are impotent in the defense against RSV. Worse doctors are still very uniformed per the butterfly effect, the entropy, that naturally occurs in the body.

In the 1960’s RSV vaccine was an unfortunate event and i am afraid one the medical community did not really learn from. When children who had received the vaccine met the wild virus during a community outbreak, they developed more sever disease, despite high levels of antibody.

Basically only one type of RSV exists; the changes in the outer coat of RSV ae so minor that the human body recognizes it as the same virous of previous years, despite the new outfit.

Several factors seem to influence the severity of RSV infection. Boys appear to be affected more severely (unsurprisingly given what we know about the fragility of males…so sensitive!). Infants that acquire their first RSV infection during the first couple of months of life are more apt to require hospitalization. RSV infection is highest in crowded heavily industrialized areas, statistically showing significant variance from more spread out affluent communities.

Considering the long term consequences it does seem reasonable to suggest that if RSV infects the lung of an infant at a particularly vulnerable stage of developmet, the lung may subsequently be more suscceptible to repeated infections and to other adverse facts such has enfiromental polluntants.

The singular epidemiological and immunological characteristics have thus far defied control.

Fortunately www.legreenpill.com does a pretty good job of finding a solution that is aligned well with traditional medince and is well proven in laboratory settings. Research is continuing in hopes to elucidate the structure and function of htis enigmatic virus, however le green pill will effectively act as a immunotherapy that functions very similar to how a vaccine works.